Primary Prostatic Mucosa Associate Lymphoid -Myassignmenthelp.Com

Question: Discuss About The Primary Prostatic Mucosa Associate Lymphoid? Answer: Introducation Emphsyema, a progressive disease of lung is caused by the innate respiratory dense mechanism of the lungs epithelial cell and mucociliary transport system after the foreign antigens infiltrate the lungs cells. The inflammatory immune cells transfer them to lymphatic tissue layer and the release of proteolytic enzymes leads to the destruction of the lungs epithelial barrier and the condition of emphysema (Goldklang Stockley, 2016). Heart rate and blood pressure are two vital signs that are affected by Mr. Browns emphysema. This occurs because the condition of emphysema results in inefficient oxygen diffusion and low oxygen content in blood. Hence, heart also suffers from lack of oxygen and compensates it by beating faster. Exacerbation of COPD results in hypertension because of airflow limitation in COPD patients (Imaizumi, Eguchi Kario, 2014). Angina pectoris, a clinical manifestation of myocardial infarction is caused by chemical and management stimulation of sensory nerve endings in the myocardium. During ischemia, the degradation of ATP to adenosine leads to the stimulation of receptors in cardiac afferent nerve ending and induces angina (Ford, Corcoran Berry, 2018). Myocardial infarction (MI) is the sudden death of myocardial tissue. The pathophysiology of the condition is linked to the thrombotic occlusion of coronary vessel caused due to rupture of plaque. It caused rapid depression of systolic function (Frangogiannis, 2015). Non-ST segment elevation myocardial infarction is one type of MI and it affects the arteries of the heart. Mr. Browns past AMI (Acute myocardial infarction) might have caused his CCF (congestive cardiac failure). This is said because heart failure complications increase in patients having AMI. It leads to acute structural complications of the heart muscle and diagnosis of CCF signifies acute alterations in left ventricular function of patient (Dharmarajan et al., 2015). Swelling of leg is a condition of accumulation of fluids in the tissue also called as edema. Mr. Browns swollen ankles and legs are related to CCF because CCF limits the ability of heart to pump blood efficiently. Hence, heart failure affect blood pumping ability of the heart and causes fluids to accumulate in different parts of the body. Benign Prostrate Hypermegaly (BPH) is a condition of enlarged prostrate in males. The prostate grows two times and the BPH results in the second growth phase. It weakens the bladder and its ability to completely empty the urinary bladder (Hashemzadeh et al., 2017). Pulmonary oedema (PO) is a clinical condition associated with accumulation of fluids in the lungs. As the condition affects a persons ability to gain oxygen, they start to struggle for breath. Hence, symptoms of running out of breath show signs of PO in patient. Nursing care plan for Mr. Brown: Care plan Patient problem One goal Course of action 1. To balance fluid intake of patient Shortness of breath, edema or swollen legs To maintain fluid balance in patient and decrease symptoms of edema Balance fluid intake in patient by means of diet changes and fluid restriction Evaluate and monitor urine output in patient 2. To manage ineffective breathing pattern in patient Shortness of breath To improve breathing rate and tissue oxygenation in patient Conduction work of breathing assessment in patient to identify severity of the issue Provide optimal body alignment to patient by placing him in semi-fowlers position 3. To help patient manage his self-care needs Activity intolerance and limited capacity for self-care in patient To help patients achieved desired activities to manage his self-care needs Provide assistance to patient in self-care activities Provide adequate exercise to patient to strengthen muscles and increase capacity for activity Allied health care workers include therapist, administrators and those staffs who supports the health care professionals in providing optimum care to patient. They provide direct care to patient and enhance the quality of life of patients (Demo et al., 2015). The person who can assist Mr. Brown after discharge includes: Nurses: The nurse can play a role is vital sign assessment of patient and providing medications on time. As Mr. Brown has symptoms of edema, she will play a role in monitoring urine output fluid intake schedule. She can also educate patients regarding the knowledge of the disease process and taking precautions in daily life. Physiotherapist: The physiotherapist can play a role in providing aerobic and resistance training to Mr. Brown. This will increase his mobility and endurance capacity (Knocke, 2012). The difference between TIA and stroke is that stroke is a permanent injury to the brain and TIA is a accounting blockage where blood flow interruption is for short time. The pathophysiology of stroke is linked to heart or blood vessel problem caused by inflammation, oxidative damage and ionic imbalances (Deb, Sharma Hassan, 2010). Fast is an acronym that helps to identify patients having strokes. The full form of FAST is: F- Facial drooping A- Arm weakness S- Speech problem T- Time to call emergency services By looking at vital observation of John, it can be said that his BP of 160/95 is a concern because this means that he is hypertensive and the condition of hypertension may further increase the chances of stroke in patients. High blood pressure is likely to damage the arteries and increase chances of stroke. Appropriate pharmacological intervention related to hypertension will be necessary then to prevent complications in patient (Appiah, Minhas Robinson, 2018). Anxiety, irregular heartbeat, rapid breathing and chest pain might be the few signs and symptoms that John might have experiences when he was diagnosed with pulmonary embolism. Dyspnea- Dyspnea is a condition of difficult breath or shortness of breath caused due to several health issues as well as due to intense exercise. Tachycardia- It is a condition of abnormally high heart rate caused due to heart related conditions like hypertension and coronary heart diseases. Haemoptysis- It is a condition leading to coughing up blood from the lungs because of bleeding within the lungs. Diaphoresis- It is a clinical condition associated with excessive sweating in a person caused due to presence of several medical conditions. Asthma is caused by the pathophysiological mechanism of airway inflammation, intermittent airflow obstruction and bronchial hyper-responsiveness. Airway inflammation is seen because of stimulation of airway smooth muscle in response to exogenous and endogenous stimuli (Sullivan et al., 2016). If John had an asthma attack, I would place him in supine position to prevent asthma symptoms as it relieves difficulty in breathing and shortness of breath (Kalolella, 2016). (Refer to the discussion section of this reference which shows that supine sleep position relieves breathing and shortness of breath in patients with asthma symptoms ) The heart rate and respiratory of John would have increased if he had an asthma attack. This is because it is caused by chronic inflammation of respiratory tubes and intermittent airflow obstruction. Such conditions increase airflow limitations thus leading to symptoms of high heart and respiratory rate. John would require the following staffs for rehabilitation following stroke: Nurse: The nurse can play a role in supporting John in activities of daily living and maintaining the continuum of care. Therapist: Therapist can help John to perform daily and demanding task like bathing, toileting and dressing. They can also provide specific training to improve the Reference: Imaizumi, Y., Eguchi, K., Kario, K. (2014). Lung Disease and Hypertension.Pulse,2(1-4), 103112. https://doi.org/10.1159/000381684 Appiah, K. O., Minhas, J. S., Robinson, T. G. (2018). Managing high blood pressure during acute ischemic stroke and intracerebral hemorrhage.Current opinion in neurology,31(1), 8-13. Deb, P., Sharma, S., Hassan, K. M. (2010). Pathophysiologic mechanisms of acute ischemic stroke: An overview with emphasis on therapeutic significance beyond thrombolysis.Pathophysiology,17(3), 197-218. Demo, D. H., Fry, D., Devine, N., Butler, A. (2015). A call for action: advocating for increased funding for the allied health professions ASAHP Leadership Development Program.Journal of allied health,44(1), 57-62. Dharmarajan, K., Hsieh, A. F., Kulkarni, V. T., Lin, Z., Ross, J. S., Horwitz, L. I., ... Krumholz, H. M. (2015). Trajectories of risk after hospitalization for heart failure, acute myocardial infarction, or pneumonia: retrospective cohort study.bmj,350, h411. Ford, T. J., Corcoran, D., Berry, C. (2018). Stable coronary syndromes: psychology, diagnostic advances and therapeutic need.Heart,104(4), 284-292. Frangogiannis, N. G. (2015). Pathophysiology of myocardial infarction.Comprehensive Physiology. Goldklang, M., Stockley, R. (2016). Pathophysiology of Emphysema and Implications.Chronic Obstructive Pulmonary Diseases,3(1), 454. Hashemzadeh, S., Farrokhi, F., Hozhabrossadaty, A., Ghafarzadegan, K., Ashraf, H. (2017). A case of recurrent hematuria in primary prostatic low grade mucosa associated lymphoid tissue.Journal of nephropathology,6(2), 49. Hsieh, C. H., Putman, K., Nichols, D., McGinty, M. E., DeJong, G., Smout, R. J., Horn, S. (2010). Physical and occupational therapy in inpatient stroke rehabilitation: The contribution of therapy extenders.American journal of physical medicine rehabilitation,89(11), 887-898. Kalolella, A. B. (2016). Sleeping position and reported night-time asthma symptoms and medication.The Pan African medical journal,24. Knocke, A. (2012). Program description: physical therapy in a heart failure clinic.Cardiopulmonary physical therapy journal,23(3), 46. Sullivan, A., Hunt, E., MacSharry, J., Murphy, D. M. (2016). The microbiome and the pathophysiology of asthma.Respiratory research,17(1), 163